MOBILE VIEW  | 

ZIKA VIRUS - MEDICAL MANAGEMENT

Classification   |    Detailed evidence-based information

Therapeutic Toxic Class

    A) Zika virus is a mosquito-borne pathogen (family Flaviviridae, genus Flavivirus) that is transmitted to humans primarily through the bite of an infected Aedes spp. mosquitoes. The infection typically produces a mild illness and many individuals remain asymptomatic. During an outbreak that started in 2015 in Brazil, microencephaly was reported in some infants born to pregnant women exposed to the Zika virus. These mosquitoes can also spread the dengue and Chikungunya viruses (see ALPHAVIRUS INFECTIONS management for further information).

Specific Substances

    1) Zika (synonym)
    2) ZIKV (synonym)
    3) Zika Infection (synonym)
    4) Zika disease (synonym)
    5) Zika virus disease (synonym)

Available Forms Sources

    A) SOURCES
    1) Zika is an arbovirus of the flavivirus genus that belongs to the flaviviridae family. Other viruses associated with this family include: dengue, yellow fever, Japanese encephalitis, West Nile and Saint-Louis encephalitis. It was originally isolated in 1947 from a rhesus monkey (Oehler et al, 2014). Zika virus is structurally similar to dengue virus; the characteristics are indistinguishable from dengue infection (Keighley et al, 2015).
    2) The first human cases of Zika virus infection were described in the 1960s in Africa followed by Southeast Asia. The Zika virus has been isolated in several African countries (Uganda, Tanzania, Egypt, Central African Republic, Sierra leone, and Gabon), Asian countries (Thailand, India, Malaysia, the Philippines, Vietnam and Indonesia) and in Micronesia. Until 2007, cases had remained sporadic until an outbreak of Zika virus in Micronesia (Oehler et al, 2014; Marcondes & Ximenes, 2015).
    3) REPORTABLE DISEASE: In 2016, the Zika virus infection became a nationally reportable disease in the United States (Centers for Disease Control and Prevention (CDC), 2016).

Life Support

    A) This overview assumes that basic life support measures have been instituted.

Clinical Effects

    0.2.1) SUMMARY OF EXPOSURE
    A) BACKGROUND: Zika is an arbovirus of the flavivirus genus that belongs to the flaviviridae family. Other viruses associated with this family include: dengue, yellow fever, Japanese encephalitis, West Nile and Saint-Louis encephalitis. It was originally isolated in 1947 from a rhesus monkey.
    B) EPIDEMIOLOGY: The first human cases were described in the 1960s in Africa followed by Southeast Asia. The Zika virus has been isolated in several African countries (Uganda, Tanzania, Egypt, Central African Republic, Sierra leone, and Gabon), Asian countries (Thailand, India, Malaysia, the Philippines, Vietnam and Indonesia) and in Micronesia. Until 2007, cases remained sporadic until an outbreak of Zika virus in Micronesia.
    C) OUTBREAKS: Prior to 2015, Zika virus infection have occurred in parts of Africa, Southeast Asia, and the Pacific Islands. In May 2015, the first confirmed cases of Zika virus infection were reported in Brazil. As of January 2016, outbreaks were occurring in many countries. The Zika virus is anticipated to continue to spread to other countries. As of July 2016, cases of Zika virus infection have been reported in a small section of Miami, Florida. Travel-related cases are likely to increase and may produce local spread of the disease in some areas of the US. Due to ongoing outbreak in many countries and the recent reports of cases within the continental United States, the CDC and state and local health departments are continuing ongoing surveillance and monitoring for the Zika virus (ie, possible primary and secondary cases) in the US.
    D) TRANSMISSION: Primarily transmitted through the bite of an infected Aedes species (genus) mosquito, which is also the same mosquito that can spread dengue and chikungunya viruses. During outbreaks, anthroponotic (human-to-vector-to human) transmission occurs. Other modes of transmission have included perinatal, in utero, and potentially blood transfusions. Sexual transmission of Zika has occurred. In February 2016, the first confirmed case of Zika virus acquired through sexual transmission was reported in the US. It has also been detected in breast milk, but transmission of the virus through breastfeeding has not been reported.
    E) VECTOR: Aedes species (genus) mosquito. For the ongoing French Polynesia epidemic, the Ae. aegypti and Ae. polynesiensis are the suspected vectors.
    F) INCUBATION PERIOD: The incubation period for the Zika virus infection is not known, but likely a few days to a week.
    G) HOST: During outbreaks of the Zika virus, humans are the primary host.
    H) INCIDENCE. In general, the incidence of Zika virus has remained unknown because it can mimic the clinical characteristics of the dengue virus unless laboratory testing is performed.
    I) WITH POISONING/EXPOSURE
    1) CLINICAL EFFECTS: Only 1 in 5 people infected with the Zika virus will become symptomatic. Of the patients that may become ill, severe disease requiring hospitalization is uncommon.
    2) MILD TO MODERATE ILLNESS: Clinical illness is usually mild and the symptoms generally include a new onset of acute fever, maculopapular rash, arthralgia, myalgia, headache or conjunctivitis. During outbreaks, other symptoms have included malaise, gastrointestinal effects, hypotension, dizziness, and axillary and inguinal lymphadenopathy.
    3) SEVERE ILLNESS: Guillain-Barre syndrome has been associated with some cases of Zika virus infection.
    4) INFANTS: During an outbreak that started in 2015 in Brazil, microencephaly was reported in some infants born to pregnant women exposed to the Zika virus. The association between pregnant women infected with the Zika virus and infants born with microencephaly or neurologic abnormalities is currently being studied.
    5) RARE: Fatalities are rare.
    0.2.3) VITAL SIGNS
    A) WITH POISONING/EXPOSURE
    1) Acute onset of fever is a common clinical finding.
    0.2.20) REPRODUCTIVE
    A) The Zika virus infection has been transmitted through both intrauterine transmission (congenital) and intrapartum transmission from a viremic pregnant woman to her newborn. It has also been detected in breast milk (Zika virus RNA); however, transmission through breastfeeding has not been reported. During the current outbreak in Brazil, an association between an increase in cases of microencephaly in infants and the Zika virus infection have occurred. Further studies are underway to the determine a possible association between the Zika virus infection and microcephaly. Sexual transmission of the Zika virus has been reported.

Laboratory Monitoring

    A) Monitor vital signs and mental status. In most cases, clinical illness is mild; however, Guillain-Barre has been reported in cases of suspected Zika virus.
    B) Any patient who presents with an acute febrile illness, rash, myalgia, or arthralgia and have travelled recently (within the past 2 weeks) where there is ongoing infection of the Zika virus or lives in an at-risk region should be tested. Testing should include Zika, chikungunya and dengue virus infections.
    C) DIAGNOSTIC EVALUATION: The diagnosis of the Zika virus is by molecular and serologic testing and includes reverse transcription-polymerase chain reaction (RT-PCR) for viral RNA, immunoglobulin M ELISA (IgM ELISA) and plaque reduction neutralization test (PRNT) for Zika antibodies. Diagnosis can vary depending on the stage of illness (ie, acute versus convalescent period). In the acute phase (first 7 days of illness), viral RNA can usually be identified in serum, and RT-PCR is preferred to test all 3 suspected types (Zika, dengue, chikungunya) of virus. In the convalescent period (serum collected more than 8 days after illness), patients should be tested by virus-specific IgM ELISA; positive results are confirmed by testing for neutralizing antibodies. For patients with a positive IgM (ie, dengue or Zika IgM ELISA), the results are considered indicative of a recent flavivirus infection; therefore, plaque-reduction neutralization (PRNT) tests should be performed to measure virus-specific neutralizing antibodies to possibly discriminate between cross-reacting antibodies in primary flavivirus infections.
    D) PREGNANT WOMEN: All pregnant women in the United States and US territories should be assessed for possible Zika virus exposure during each prenatal visit. Pregnant women who report signs or symptoms consistent with Zika virus exposure (ie, acute onset of fever, rash, arthralgia, conjunctivitis) should be tested for the virus.
    1) SYMPTOMATIC PREGNANT WOMEN: rRT-PCR testing of serum and urine should be performed in women that seek care less than 2 weeks after symptom onset. A positive result confirms the diagnosis of recent maternal Zika virus infection. Symptomatic pregnant women with a negative result should have Zika virus IgM and dengue virus IgM antibody testing performed. If either test are positive or equivocal results, PRNT should be performed on the same IgM-tested sample to rule out a false-positive result.
    2) Symptomatic pregnant women who seek care 2 to 12 weeks after symptom onset should initially have Zika virus and dengue virus IgM antibody testing done.
    3) In symptomatic and asymptomatic pregnant women with possible Zika virus exposure who seek care greater than 12 weeks after symptom onset or possible exposure, IgM antibody testing should be considered. Because of the limitations of testing beyond 12 weeks, serial fetal ultrasounds should be considered as indicated.
    4) ASYMPTOMATIC PREGNANT WOMEN: Testing of asymptomatic pregnant women differs depending on the possible circumstances of exposure. Asymptomatic and NOT living in an area with active Zika virus transmission less than 2 weeks after possible exposure: Perform rRT-PCR (serum and urine) testing. Asymptomatic and NOT living in an area with active Zika virus transmission 2 to 12 weeks after possible exposure OR asymptomatic and living in an area with active Zika transmission (includes first and second trimester): Obtain Zika and dengue IgM (serum).
    5) PRENATAL MONITORING OF POSITIVE/PROBABLE CASES: RECENT ZIKA VIRUS INFECTION: Consider serial ultrasounds every 3 to 4 weeks to follow fetal anatomy and growth. Amniocentesis is based on individual clinical circumstances. PRESUMPTIVE RECENT ZIKA VIRUS INFECTION: Consider serial ultrasounds every 3 to 4 weeks to follow fetal anatomy and growth. Amniocentesis is based on individual clinical circumstances. NO EVIDENCE OF ZIKA VIRUS OR DENGUE VIRUS INFECTION: Prenatal ultrasound to evaluate for fetal abnormalities consistent with congenital Zika virus syndrome. If fetal abnormalities are present, repeat Zika virus rRT-PCR and IgM testing. Clinical management should be based on laboratory results.
    E) INFANTS: Due to concerns about microencephaly associated with maternal exposure to the Zika virus, fetuses and infants of women infected with the virus should be evaluated for congenital exposure and possible neurologic abnormalities. Currently, molecular and serologic testing are recommended by the CDC to diagnose congenital infection because of the uncertainty in determining which method is most reliable in this age group.
    F) Monitor fluid status and electrolytes in patients that develop significant vomiting and/or diarrhea.
    G) No specific laboratory studies are needed for most patients that develop mild clinical symptoms. Obtain a baseline CBC in patients as indicated; the presence of neutropenia, thrombocytopenia and hemorrhage are not typical of Zika virus, but are more likely to occur with dengue virus infection.
    H) Beginning in 2016, the US FDA has issued an Emergency Use Authorization for several diagnostic tools that can be used for testing for the Zika virus. They include Trioplex Real Time RT-PCR assay and the Zika MAC-ELISA, which have been distributed to qualified laboratories in the US.
    I) BLOOD DONATION: As of late August, 2016 the US FDA has recommended that all blood donations (blood and blood components) collected in the US and US territories be tested for the Zika virus using an investigational individual donor nucleic acid test (ID-NAT). Donors that are reactive for Zika virus should be deferred from donating for 120 days from the date of the reactive test. Donors should also be counseled regarding a possible exposure/infection.

Treatment Overview

    0.4.7) BITES/STINGS
    A) MANAGEMENT OF MILD TO MODERATE TOXICITY
    1) Treatment is symptomatic and supportive. There is no specific antiviral therapy for Zika virus infection. Acute symptoms usually resolve in a few days to a week. Monitor vital signs and mental status. Treat symptoms with fluids, rest, analgesics and antipyretics. Aspirin and other nonsteroidal anti-inflammatory drugs (NSAIDs) should be avoided until dengue fever has been ruled out due to the potential risk for hemorrhage. Monitor serum electrolytes if the patient develops significant vomiting and/or diarrhea. Replace volume loss with oral or IV fluids as needed. Manage mild hypotension with IV fluids.
    B) MANAGEMENT OF SEVERE TOXICITY
    1) Treatment is symptomatic and supportive. Monitor vital signs and mental status. Treat severe hypotension with IV 0.9% NaCl at 10 to 20 mL/kg. Add dopamine, norepinephrine or pressor of choice if unresponsive to fluids.
    C) PREVENTION
    1) SUMMARY: The most important thing to prevent the Zika infection and other related illnesses (ie, dengue and chikungunya) is to avoid mosquito bites. In areas where outbreaks have occurred, individuals should avoid mosquito bites by wearing long sleeve shirts and pants, staying indoors when possible, use of bed nets in areas where air conditioning is not available, use of insect repellents containing DEET, picardin, and IR3535 (follow label directions), and protect infants and children similar to adults, except insect repellent should NOT be used on infants younger than 2 months of age.
    2) PROTECTING OTHERS FROM INFECTION: If a patient has the Zika virus infection, it is important for that individual to avoid mosquito bites during the first week of illness. The Zika virus can be found in the blood of an infected individual during the first week of infection.
    3) PREVENTION OF SEXUAL TRANSMISSION: Sexual transmission of Zika has been reported. Abstinence or the consistent use of barrier methods (condoms) can reduce the risk of transmission. Due to the potential risk of maternal Zika virus infection, its suggested that pregnant women whose partners have or are at risk for the infection use barrier methods against the transmission of the virus or abstain from sexual intercourse.
    D) DECONTAMINATION
    1) PREHOSPITAL: Prehospital GI decontamination including activated charcoal is not indicated. The focus of prehospital care should include supportive care.
    2) HOSPITAL: GI decontamination is not indicated. HEALTH CARE WORKERS: Based on the evolving nature of this outbreak in some areas, barrier techniques and infection control (avoid direct contact with blood or bodily fluids) are recommended while treating any patient with confirmed or suspected Zika virus infection.
    E) ANTIDOTE
    1) No antidote or vaccine is available for human exposure to Zika virus. Currently, research is underway to develop a vaccine to prevent Zika virus infection.
    F) AIRWAY MANAGEMENT
    1) Zika virus typically results in cases of mild, self-limiting illness; airway support is unlikely to be necessary. If airway compromise occurs, supportive measures including endotracheal intubation and mechanical ventilation may be necessary in patients that develop neurologic complications.
    G) ENHANCED ELIMINATION
    1) Enhanced elimination is not indicated in patients that develop Zika virus infection.
    H) PATIENT DISPOSITION
    1) HOME CRITERIA: Asymptomatic patients or patients with minimal symptoms may remain at home.
    2) OBSERVATION CRITERIA: Patients with worsening symptoms that do not improve with over-the-counter medications (eg, analgesics, antipyretics) and basic home treatments should go to a healthcare facility for evaluation and treatment. Patients who are stable with improving symptoms may be sent home.
    3) ADMISSION CRITERIA: Hospital admission is uncommon; however, unstable patients or those with worsening symptoms should be admitted to the hospital. Those with severe symptoms (eg, neurologic abnormalities, Guillain-Barre syndrome) may require an ICU admission. Criteria for discharge include patients that are clinically improving and stable.
    4) CONSULT CRITERIA: Infectious disease physicians and/or intensivists may be consulted for medical advice. Pregnant women who have tested positive for the Zika virus should be referred to an infectious disease specialist with expertise in prenatal management. Rheumatologists should be consulted in patients that develop severe joint swelling, arthritis and/or polyarthritis symptoms. Poison centers can aid treatment by serving as a public health resource by providing advice to the general public, and working with local and regional health departments.
    I) DIFFERENTIAL DIAGNOSIS
    1) In endemic areas, the Zika virus may appear similar to dengue fever or chikungunya illness. Because of ifs similar clinical features with other infection, the following should be considered leptospirosis, malaria, rickettsia, group A streptococcus, rubella, measles, and parvovirus, enterovirus, adenovirus, and other alphavirus infections (eg, Mayaro, Ross River, Barmah Forest, O'nyong-nyong and Sindbis viruses).

Range Of Toxicity

    A) TOXICITY: The Zika virus is primarily transmitted by the bite of an infected Aedes aegypti mosquitoes which is the same mosquito that can spread dengue and chikungunya viruses. During outbreaks of the Zika virus, anthroponotic (human-to-vector-to human) transmission occurs. Other modes of transmission have included perinatal, in utero, and potentially blood transfusions and sexual contact with an infected partner. The virus typically results in no symptoms or a mild self-limited illness. A specific viral dose is unknown. Fatalities are rare, but microencephaly has occurred in infants during the recent outbreak of the virus in Brazil. Other potentially serious adverse effects have been sporadic reports of Guillain-Barre syndrome in adults.

Summary Of Exposure

    A) BACKGROUND: Zika is an arbovirus of the flavivirus genus that belongs to the flaviviridae family. Other viruses associated with this family include: dengue, yellow fever, Japanese encephalitis, West Nile and Saint-Louis encephalitis. It was originally isolated in 1947 from a rhesus monkey.
    B) EPIDEMIOLOGY: The first human cases were described in the 1960s in Africa followed by Southeast Asia. The Zika virus has been isolated in several African countries (Uganda, Tanzania, Egypt, Central African Republic, Sierra leone, and Gabon), Asian countries (Thailand, India, Malaysia, the Philippines, Vietnam and Indonesia) and in Micronesia. Until 2007, cases remained sporadic until an outbreak of Zika virus in Micronesia.
    C) OUTBREAKS: Prior to 2015, Zika virus infection have occurred in parts of Africa, Southeast Asia, and the Pacific Islands. In May 2015, the first confirmed cases of Zika virus infection were reported in Brazil. As of January 2016, outbreaks were occurring in many countries. The Zika virus is anticipated to continue to spread to other countries. As of July 2016, cases of Zika virus infection have been reported in a small section of Miami, Florida. Travel-related cases are likely to increase and may produce local spread of the disease in some areas of the US. Due to ongoing outbreak in many countries and the recent reports of cases within the continental United States, the CDC and state and local health departments are continuing ongoing surveillance and monitoring for the Zika virus (ie, possible primary and secondary cases) in the US.
    D) TRANSMISSION: Primarily transmitted through the bite of an infected Aedes species (genus) mosquito, which is also the same mosquito that can spread dengue and chikungunya viruses. During outbreaks, anthroponotic (human-to-vector-to human) transmission occurs. Other modes of transmission have included perinatal, in utero, and potentially blood transfusions. Sexual transmission of Zika has occurred. In February 2016, the first confirmed case of Zika virus acquired through sexual transmission was reported in the US. It has also been detected in breast milk, but transmission of the virus through breastfeeding has not been reported.
    E) VECTOR: Aedes species (genus) mosquito. For the ongoing French Polynesia epidemic, the Ae. aegypti and Ae. polynesiensis are the suspected vectors.
    F) INCUBATION PERIOD: The incubation period for the Zika virus infection is not known, but likely a few days to a week.
    G) HOST: During outbreaks of the Zika virus, humans are the primary host.
    H) INCIDENCE. In general, the incidence of Zika virus has remained unknown because it can mimic the clinical characteristics of the dengue virus unless laboratory testing is performed.
    I) WITH POISONING/EXPOSURE
    1) CLINICAL EFFECTS: Only 1 in 5 people infected with the Zika virus will become symptomatic. Of the patients that may become ill, severe disease requiring hospitalization is uncommon.
    2) MILD TO MODERATE ILLNESS: Clinical illness is usually mild and the symptoms generally include a new onset of acute fever, maculopapular rash, arthralgia, myalgia, headache or conjunctivitis. During outbreaks, other symptoms have included malaise, gastrointestinal effects, hypotension, dizziness, and axillary and inguinal lymphadenopathy.
    3) SEVERE ILLNESS: Guillain-Barre syndrome has been associated with some cases of Zika virus infection.
    4) INFANTS: During an outbreak that started in 2015 in Brazil, microencephaly was reported in some infants born to pregnant women exposed to the Zika virus. The association between pregnant women infected with the Zika virus and infants born with microencephaly or neurologic abnormalities is currently being studied.
    5) RARE: Fatalities are rare.

Vital Signs

    3.3.1) SUMMARY
    A) WITH POISONING/EXPOSURE
    1) Acute onset of fever is a common clinical finding.
    3.3.3) TEMPERATURE
    A) WITH POISONING/EXPOSURE
    1) Acute onset of fever is a common clinical finding of Zika virus infection (Centers for Disease Control and Prevention (CDC), 2016).

Heent

    3.4.3) EYES
    A) WITH POISONING/EXPOSURE
    1) Conjunctivitis is a common clinical finding of Zika virus infection (Centers for Disease Control and Prevention (CDC), 2016). Retro-orbital pain has also been reported (Keighley et al, 2015).

Cardiovascular

    3.5.2) CLINICAL EFFECTS
    A) HYPOTENSIVE EPISODE
    1) WITH POISONING/EXPOSURE
    a) CASE REPORTS: In Indonesia, 7 patients that were serologically reactive to the Zika virus developed high fever, chills, malaise, anorexia, vomiting, diarrhea, abdominal pain, dizziness, leg pain, lymphadenopathy and hypotension (Marcondes & Ximenes, 2015).

Neurologic

    3.7.2) CLINICAL EFFECTS
    A) HEADACHE
    1) WITH POISONING/EXPOSURE
    a) Headache is a common clinical finding of Zika virus infection (Centers for Disease Control and Prevention (CDC), 2016). Retroocular headaches have also been reported (Oehler et al, 2014)
    B) DIZZINESS
    1) WITH POISONING/EXPOSURE
    a) CASE REPORTS: In Indonesia, 7 patients that were serologically reactive to the Zika virus developed high fever, chills, malaise, anorexia, vomiting, diarrhea, abdominal pain, dizziness, leg pain, lymphadenopathy and hypotension (Marcondes & Ximenes, 2015).
    C) GUILLAIN-BARRĆ© SYNDROME
    1) WITH POISONING/EXPOSURE
    a) Guillain-Barre syndrome has been reported in some patients following suspected Zika virus infection (Centers for Disease Control and Prevention (CDC), 2016).
    b) CASE REPORT: In French Polynesia, a woman with a history of acute articular rheumatism developed Guillain-Barre syndrome following an episode of flu-like illness (myalgia, fever, cutaneous rash and conjunctivitis) consistent with Zika virus infection. She was admitted with paresthesia and ascendant muscular weakness. During her hospitalization, she developed tetraparesis that was predominant in the lower limbs, with paresthesia of the extremities, diffuse myalgia and a bilateral but asymmetric peripheral facial palsy. Deep tendon reflexes were absent. Respiratory and swallowing were normal. An electromyogram showed signs of a diffuse demyelinating disorder. Symptoms improved with the administration of polyvalent immunoglobulin. Paraparesis persisted requiring a walker for ambulation at the time of discharge. By day 40, she was able to walk independently and had normal muscle strength. Of note, an epidemic of Zika virus infection is ongoing in this region (Oehler et al, 2014).
    1) Following the report of this case, another 40 cases of Guillain-Barre syndrome were reported in this same country (Marcondes & Ximenes, 2015).
    D) MICROCEPHALUS
    1) WITH POISONING/EXPOSURE
    a) The Zika virus has been confirmed in infants with microcephaly. During an outbreak that started in 2015 in Brazil, an increased number of infants born with microcephaly has been reported; however, the number of microcephaly cases associated with the Zika virus remains unknown (Petersen et al, 2016; Staples et al, 2016). Testing was positive for the virus in some infants with microcephaly and in some women who experienced fetal loss. The virus was detected in various specimens including brain tissue, the placenta and amniotic fluid (Staples et al, 2016).
    b) According to the CDC, further studies are underway to determine the association between Zika virus infection and microcephaly including any contributory factors (ie, environment, nutrition, concurrent infections) (Petersen et al, 2016).

Gastrointestinal

    3.8.2) CLINICAL EFFECTS
    A) GASTROINTESTINAL TRACT FINDING
    1) WITH POISONING/EXPOSURE
    a) CASE REPORTS: In Indonesia, 7 patients that were serologically reactive to the Zika virus developed high fever, chills, malaise, anorexia, vomiting, diarrhea, abdominal pain, dizziness, leg pain, lymphadenopathy and hypotension (Marcondes & Ximenes, 2015).

Dermatologic

    3.14.2) CLINICAL EFFECTS
    A) ERUPTION
    1) WITH POISONING/EXPOSURE
    a) Maculopapular rash is a common clinical finding of Zika virus infection (Centers for Disease Control and Prevention (CDC), 2016).

Musculoskeletal

    3.15.2) CLINICAL EFFECTS
    A) JOINT PAIN
    1) WITH POISONING/EXPOSURE
    a) Arthralgia is a common clinical finding of Zika virus infection (Centers for Disease Control and Prevention (CDC), 2016).
    B) MUSCLE PAIN
    1) WITH POISONING/EXPOSURE
    a) Myalgia is also likely to occur in individuals with the Zika virus infection (Centers for Disease Control and Prevention (CDC), 2016).

Immunologic

    3.19.2) CLINICAL EFFECTS
    A) LYMPHADENOPATHY
    1) WITH POISONING/EXPOSURE
    a) CASE REPORTS: In Indonesia, 7 patients that were serologically reactive to the Zika virus developed high fever, chills, malaise, anorexia, vomiting, diarrhea, abdominal pain, dizziness, leg pain, lymphadenopathy and hypotension (Marcondes & Ximenes, 2015).

Reproductive

    3.20.1) SUMMARY
    A) The Zika virus infection has been transmitted through both intrauterine transmission (congenital) and intrapartum transmission from a viremic pregnant woman to her newborn. It has also been detected in breast milk (Zika virus RNA); however, transmission through breastfeeding has not been reported. During the current outbreak in Brazil, an association between an increase in cases of microencephaly in infants and the Zika virus infection have occurred. Further studies are underway to the determine a possible association between the Zika virus infection and microcephaly. Sexual transmission of the Zika virus has been reported.
    3.20.3) EFFECTS IN PREGNANCY
    A) MICROENCEPHALY
    1) The Zika virus has been confirmed in infants with microcephaly. During an outbreak that started in 2015 in Brazil, an increased number of infants born with microcephaly has been reported; however, the number of microcephaly cases associated with the Zika virus remains unknown (Petersen et al, 2016). Testing was positive for the virus in some infants with microcephaly and in some women who experienced fetal loss. The virus was detected in various specimens including brain tissue, the placenta and amniotic fluid (Staples et al, 2016).
    2) According to the CDC, further studies are underway to determine the association between Zika virus infection and microcephaly including any contributory factors (ie, environment, nutrition, concurrent infections) (Petersen et al, 2016).
    B) MODE OF TRANSMISSION
    1) At the time of this review, the Zika virus infection has been transmitted through both intrauterine transmission (congenital) and intrapartum transmission from a viremic pregnant woman to her newborn. In Brazil, the virus has been detected in brain tissue, the placenta and amniotic fluid of some infants diagnosed with microencephaly and from infected pregnant women who experienced fetal loss (Staples et al, 2016).
    2) Maternal-fetal transmission of the virus appears to occur throughout pregnancy (Petersen et al, 2016).
    3) Sexual transmission of the Zika virus has been reported (Centers for Disease Control and Prevention (CDC), 2016e).
    a) In February 2016, the first confirmed case of Zika virus acquired through sexual transmission was reported in Dallas. The patient was infected with the virus after having sexual contact with an ill partner who had returned from a country where the Zika virus was present (Dallas County Department of Health and Human Services, 2016). As of July 2016, 15 cases of Zika virus infection through sexual transmission have been reported in the United States (Brooks et al, 2016).
    b) PREVENTION OF SEXUAL TRANSMISSION: Sexual transmission of Zika has been reported. Sexual exposure includes vaginal, anal, oral sex or contact with genital secretions. To prevent sexual transmission of Zika virus, couples who meet any of the following criteria may consider abstaining from sex or using barrier methods against transmission of the virus (Brooks et al, 2016):
    1) A male partner with a confirmed Zika virus infection or clinical illness consistent with Zika virus disease: at least 6 months after onset of illness.
    2) A female partner with a confirmed Zika virus infection or clinical illness consisted with Zika virus disease: at least 8 weeks after onset of illness.
    3) Couples who live in areas without active Zika transmission but with 1 partner who traveled to or lived in an area of active transmission but did not develop symptoms: at least 8 weeks after that partner departed the Zika-affected area.
    4) Couples who live in an area of active Zika virus transmission: while active transmission persists.
    5) Testing of specimens to assess risk for sexual transmission is not recommended.
    C) FUTURE PREGNANCIES
    1) According to the CDC, the Zika virus infection does not pose a risk of birth defects for future pregnancies. The virus usually remains in the blood for about a week in individuals with the infection. Once the virus is cleared from the blood of a woman, it will not cause infection in an infant conceived after that time (Centers for Disease Control and Prevention (CDC), 2016).
    3.20.4) EFFECTS DURING BREAST-FEEDING
    A) BREAST MILK
    1) The Zika virus has been detected in breast milk (Zika virus RNA); however, transmission through breastfeeding has not been reported (Staples et al, 2016).

Monitoring Parameters Levels

    4.1.1) SUMMARY
    A) Monitor vital signs and mental status. In most cases, clinical illness is mild; however, Guillain-Barre has been reported in cases of suspected Zika virus.
    B) Any patient who presents with an acute febrile illness, rash, myalgia, or arthralgia and have travelled recently (within the past 2 weeks) where there is ongoing infection of the Zika virus or lives in an at-risk region should be tested. Testing should include Zika, chikungunya and dengue virus infections.
    C) DIAGNOSTIC EVALUATION: The diagnosis of the Zika virus is by molecular and serologic testing and includes reverse transcription-polymerase chain reaction (RT-PCR) for viral RNA, immunoglobulin M ELISA (IgM ELISA) and plaque reduction neutralization test (PRNT) for Zika antibodies. Diagnosis can vary depending on the stage of illness (ie, acute versus convalescent period). In the acute phase (first 7 days of illness), viral RNA can usually be identified in serum, and RT-PCR is preferred to test all 3 suspected types (Zika, dengue, chikungunya) of virus. In the convalescent period (serum collected more than 8 days after illness), patients should be tested by virus-specific IgM ELISA; positive results are confirmed by testing for neutralizing antibodies. For patients with a positive IgM (ie, dengue or Zika IgM ELISA), the results are considered indicative of a recent flavivirus infection; therefore, plaque-reduction neutralization (PRNT) tests should be performed to measure virus-specific neutralizing antibodies to possibly discriminate between cross-reacting antibodies in primary flavivirus infections.
    D) PREGNANT WOMEN: All pregnant women in the United States and US territories should be assessed for possible Zika virus exposure during each prenatal visit. Pregnant women who report signs or symptoms consistent with Zika virus exposure (ie, acute onset of fever, rash, arthralgia, conjunctivitis) should be tested for the virus.
    1) SYMPTOMATIC PREGNANT WOMEN: rRT-PCR testing of serum and urine should be performed in women that seek care less than 2 weeks after symptom onset. A positive result confirms the diagnosis of recent maternal Zika virus infection. Symptomatic pregnant women with a negative result should have Zika virus IgM and dengue virus IgM antibody testing performed. If either test are positive or equivocal results, PRNT should be performed on the same IgM-tested sample to rule out a false-positive result.
    2) Symptomatic pregnant women who seek care 2 to 12 weeks after symptom onset should initially have Zika virus and dengue virus IgM antibody testing done.
    3) In symptomatic and asymptomatic pregnant women with possible Zika virus exposure who seek care greater than 12 weeks after symptom onset or possible exposure, IgM antibody testing should be considered. Because of the limitations of testing beyond 12 weeks, serial fetal ultrasounds should be considered as indicated.
    4) ASYMPTOMATIC PREGNANT WOMEN: Testing of asymptomatic pregnant women differs depending on the possible circumstances of exposure. Asymptomatic and NOT living in an area with active Zika virus transmission less than 2 weeks after possible exposure: Perform rRT-PCR (serum and urine) testing. Asymptomatic and NOT living in an area with active Zika virus transmission 2 to 12 weeks after possible exposure OR asymptomatic and living in an area with active Zika transmission (includes first and second trimester): Obtain Zika and dengue IgM (serum).
    5) PRENATAL MONITORING OF POSITIVE/PROBABLE CASES: RECENT ZIKA VIRUS INFECTION: Consider serial ultrasounds every 3 to 4 weeks to follow fetal anatomy and growth. Amniocentesis is based on individual clinical circumstances. PRESUMPTIVE RECENT ZIKA VIRUS INFECTION: Consider serial ultrasounds every 3 to 4 weeks to follow fetal anatomy and growth. Amniocentesis is based on individual clinical circumstances. NO EVIDENCE OF ZIKA VIRUS OR DENGUE VIRUS INFECTION: Prenatal ultrasound to evaluate for fetal abnormalities consistent with congenital Zika virus syndrome. If fetal abnormalities are present, repeat Zika virus rRT-PCR and IgM testing. Clinical management should be based on laboratory results.
    E) INFANTS: Due to concerns about microencephaly associated with maternal exposure to the Zika virus, fetuses and infants of women infected with the virus should be evaluated for congenital exposure and possible neurologic abnormalities. Currently, molecular and serologic testing are recommended by the CDC to diagnose congenital infection because of the uncertainty in determining which method is most reliable in this age group.
    F) Monitor fluid status and electrolytes in patients that develop significant vomiting and/or diarrhea.
    G) No specific laboratory studies are needed for most patients that develop mild clinical symptoms. Obtain a baseline CBC in patients as indicated; the presence of neutropenia, thrombocytopenia and hemorrhage are not typical of Zika virus, but are more likely to occur with dengue virus infection.
    H) Beginning in 2016, the US FDA has issued an Emergency Use Authorization for several diagnostic tools that can be used for testing for the Zika virus. They include Trioplex Real Time RT-PCR assay and the Zika MAC-ELISA, which have been distributed to qualified laboratories in the US.
    I) BLOOD DONATION: As of late August, 2016 the US FDA has recommended that all blood donations (blood and blood components) collected in the US and US territories be tested for the Zika virus using an investigational individual donor nucleic acid test (ID-NAT). Donors that are reactive for Zika virus should be deferred from donating for 120 days from the date of the reactive test. Donors should also be counseled regarding a possible exposure/infection.
    4.1.2) SERUM/BLOOD
    A) No specific laboratory studies are needed for most patients that develop mild clinical symptoms. Obtain a baseline CBC in patients as indicated; the presence of neutropenia, thrombocytopenia and hemorrhage are not typical of Zika virus, but are more likely to occur with dengue virus infection.
    4.1.3) URINE
    A) In one study, the Zika virus was detected in urine samples using the ZIKV RNA study; samples were positive for greater than 10 days after the onset of disease (Gourinat et al, 2015).

Methods

    A) GENERAL GUIDELINES
    1) SUMMARY: Transmission of multiple arborviruses in countries in the Americas can produce febrile illness with rash, myalgia, or arthralgia; therefore, laboratory testing should confirm the etiology of these diseases (ie, Zika, chikungunya and dengue virus). Laboratory testing usually includes serum testing to detect viral nucleic acid or virus-specific immunoglobulin (Ig) M and neutralizing antibodies. It has been determined that serological cross-reactivity is possible between Zika and dengue viruses so molecular detection is necessary in acute specimens (Centers for Disease Control and Prevention (CDC), 2016). It is anticipated that locally acquired mosquito borne cases will occur in some areas of the United States.
    2) WHO SHOULD BE TESTED: Any patient who presents with an acute febrile illness, rash, myalgia, or arthralgia and have travelled recently (within the past 2 weeks) where there is ongoing infection of the Zika virus. Testing should include Zika, chikungunya and dengue virus infections (Centers for Disease Control and Prevention (CDC), 2016).
    3) REPORTABLE DISEASE: As an arboviral disease, Zika virus infection is a nationally reportable disease (Staples et al, 2016).
    4) LABORATORY SAFETY: Zika and dengue viruses are classified as biological safety level (BSL) 2 pathogens and chikungunya virus is classified as a BSL 3 agent and all should be handled appropriately with Biosafety in Microbiological and Biomedical Laboratories (BMBL) guidelines (Centers for Disease Control and Prevention (CDC), 2016).
    5) DIAGNOSTIC TESTING: Beginning in 2016, the US FDA has issued an Emergency Use Authorization for several diagnostic tools that can be used for testing for the Zika virus. They include Trioplex Real Time RT-PCR assay and the Zika MAC-ELISA, which have been distributed to qualified laboratories in the US (Rabe et al, 2016).
    6) BLOOD DONATION: As of late August, 2016 the US FDA has recommended that all blood donations (blood and blood components) collected in the US and US territories be tested for the Zika virus using an investigational individual donor nucleic acid test (ID-NAT). Donors that are reactive for Zika virus should be deferred from donating for 120 days from the date of the reactive test. Donors should also be counseled regarding a possible exposure/infection (http://www.fda.gove/downloads/biologiicsBloodvaccines/GuidanceComplianceRegulatoryInformation/Guidances/Blood/UCM518213.pdf).
    a) Donation centers in Florida and Puerto Rico should begin testing immediately.
    b) Donation centers in the following states: Alabama, Arizona, California, Georgia, Hawaii, Louisiana, Mississippi, New Mexico, New York, South Carolina and Texas should implement these recommendations as soon as possible but not later than 4 weeks after the guidance issue date (8/26/2016).
    c) Donation centers in all other states and territories should implement the recommendations as soon as possible, but not later than 12 weeks after the issue date.
    B) STAGE OF ILLNESS
    1) Diagnosis of Zika virus can vary depending on the stage of illness (acute versus convalescent period). In the acute phase (first 7 days of illness), viral RNA can usually be identified in serum, and RT-PCR is preferred to test all 3 types (Zika, dengue, chikungunya) of viruses. In the convalescent period (serum collected more than 8 days after illness), patients should be tested by virus-specific IgM ELISA; positive results are confirmed by testing for neutralizing antibodies. For patients with a positive IgM (ie, dengue or Zika IgM ELISA), the results are considered indicative of a recent flavivirus infection; therefore, plaque-reduction neutralization tests should be performed to measure virus-specific neutralizing antibodies to possibly discriminate between cross-reacting antibodies in primary flavivirus infections (Centers for Disease Control and Prevention (CDC), 2016).
    a) In patients with a primary flavivirus infection, a 4-fold or greater increase in virus-specific neutralizing antibodies between the acute- and convalescent phase serum specimens obtained 2 to 3 weeks apart can be used to confirm a recent infection (Centers for Disease Control and Prevention (CDC), 2016).
    b) In patients previously immunized against (eg, Yellow fever or Japanese encephalitis vaccine) or previously infected with another flavivirus infection (eg, West Nile, St. Louis encephalitis virus), cross-reactive antibodies (ie, IgM, neutralizing antibody assays) make it difficult to diagnose the patient's current illness (Centers for Disease Control and Prevention (CDC), 2016).
    C) SPECIMEN TESTING AND COLLECTION
    1) Zika, chikungunya and dengue virus testing can be performed at the Centers for Disease Control (CDC) and some state health departments that already perform testing for the West Nile virus RT-PCR assay (Centers for Disease Control and Prevention (CDC), 2016; Centers for Disease Control and Prevention (CDC), 2016). Tests performed at the CDC include Zika, chikungunya and dengue virus RT-PCR, IgM ELISA, and plaque reduction neutralization tests (PRNT). If testing is needed, contact your state health department for further information (Centers for Disease Control and Prevention (CDC), 2016).
    2) Commercial diagnostic assays or testing kits for the Zika virus are NOT currently available (Centers for Disease Control and Prevention (CDC), 2016).
    3) Results are usually available 4 to 14 days after receipt of the specimen. Once testing is completed, ALL results will be sent to the appropriate state health department. In addition, state health departments should be notified of any direct submissions to the CDC (Centers for Disease Control and Prevention (CDC), 2016)
    4) For additional assistance in collecting or sending laboratory samples to CDC, contact the Arboviral Diseases Branch on-call epidemiologist at 970-221-6400 or contact them at http://www.cdc.gov/ncezid/dvbd/specimensub/arboviral-shipping.html (Centers for Disease Control and Prevention (CDC), 2016).
    5) Within Puerto Rico, please call 787-706-2399 for further questions about testing (Centers for Disease Control and Prevention (CDC), 2016).
    D) PREGNANT WOMEN
    1) All pregnant women in the United States and US territories should be assessed for possible Zika virus exposure during each prenatal visit (Oduyebo et al, 2016).
    2) CDC recommends that pregnant women not travel to an area with active Zika virus transmission. Pregnant women who must travel to one of these areas should strictly follow steps to prevent mosquito bites (Oduyebo et al, 2016).
    3) SYMPTOMATIC PREGNANT WOMEN
    a) Pregnant women who report signs or symptoms consistent with Zika virus exposure (ie, acute onset of fever, rash, arthralgia, conjunctivitis) should be tested for the virus (Oduyebo et al, 2016).
    b) Maternal testing includes the same studies as the general population (Petersen et al, 2016); however, the type of testing recommended can vary depending on the time of evaluation relative to symptom onset (Oduyebo et al, 2016).
    1) rRT-PCR testing of serum and urine should be performed in women that seek care less than 2 weeks after symptom onset. A positive result confirms the diagnosis of recent maternal Zika virus infection (Oduyebo et al, 2016).
    2) Symptomatic pregnant women with a negative result should have Zika virus IgM and dengue virus IgM antibody testing performed. If either test are positive or equivocal results, PRNT should be performed on the same IgM-tested sample to rule out a false-positive result (Oduyebo et al, 2016).
    3) Symptomatic pregnant women who seek care 2 to 12 weeks after symptom onset should initially have Zika virus and dengue virus IgM antibody testing done. Positive or equivocal results require rRT-PCR testing that should be performed on the same sample to evaluate whether Zika virus RNA is present; a positive result confirms recent maternal Zika virus infection. However, if the rRT-PCR is negative and there is a positive or equivocal Zika virus IgM antibody, a PRNT should be performed. Likewise, if a patient has a positive or equivocal dengue IgM antibody test result with a negative Zika virus IgM antibody, the test should also be confirmed by PRNT (Oduyebo et al, 2016).
    4) SYMPTOMATIC AND ASYMPTOMATIC PREGNANT WOMEN WHO SEEK CARE AFTER 12 WEEKS
    a) In symptomatic and asymptomatic pregnant women with possible Zika virus exposure who seek care greater than 12 weeks after symptom onset or possible exposure, IgM antibody testing should be considered. If there is evidence of fetal abnormalities, rRT-PCR testing should be done on maternal serum and urine. However, a negative IgM antibody or rRT-PCR obtained greater than 12 weeks after symptoms does NOT rule out recent Zika virus infection. Because of the limitations of testing beyond 12 weeks, serial fetal ultrasounds should be considered (Oduyebo et al, 2016)
    5) ASYMPTOMATIC PREGNANT WOMEN
    a) Testing of asymptomatic pregnant women differs depending on the possible circumstances of exposure (Oduyebo et al, 2016):
    1) Asymptomatic and NOT living in an area with active Zika virus transmission less than 2 weeks after possible exposure: Perform rRT-PCR (serum and urine) testing (Oduyebo et al, 2016):
    1) If negative, obtain Zika virus IgM antibody testing 2 to 12 weeks after possible exposure. If both, Zika virus and dengue virus IgM are negative - No recent Zika virus infection (Oduyebo et al, 2016).
    2) Asymptomatic and NOT living in an area with active Zika virus transmission 2 to 12 weeks after possible exposure OR asymptomatic and living in an area with active Zika transmission (includes first and second trimester): Obtain Zika and dengue IgM (serum) (Oduyebo et al, 2016).
    1) If Zika virus IgM positive or equivocal and any results on dengue virus IgM: Presumptive recent Zika or flavivirus infection. Follow with rRT-PCR testing (serum); a positive result indicates recent Zika virus infection. A negative finding requires PRNT testing. The test results are as follows (Oduyebo et al, 2016):
    a) A Zika virus PRNT greater or equal to 10 and dengue virus PRNT of less than 10: Recent Zika virus infection.
    b) A Zika virus PRNT greater or equal to 10 and dengue virus PRNT greater or equal to 10: Recent flavivirus infection, however, a specific virus cannot be identified.
    c) A Zika virus PRNT of less than 10: NO recent evidence of Zika virus infection.
    2) If Zika and dengue IgM are negative: No recent Zika virus infection (Oduyebo et al, 2016).
    3) If dengue virus IgM positive or equivocal and Zika IgM virus is negative: Presumptive dengue virus infection. Obtain PRNT to confirm findings (Oduyebo et al, 2016).
    6) PRENATAL MONITORING OF PREGNANT WOMEN
    a) POSITIVE LABORATORY EVIDENCE OF CONFIRMED OR POSSIBLE CASES
    1) RECENT ZIKA VIRUS INFECTION: Consider serial ultrasounds every 3 to 4 weeks to follow fetal anatomy and growth. Amniocentesis is based on individual clinical circumstances (Oduyebo et al, 2016).
    2) PRESUMPTIVE RECENT ZIKA VIRUS INFECTION: Consider serial ultrasounds every 3 to 4 weeks to follow fetal anatomy and growth. Amniocentesis is based on individual clinical circumstances (Oduyebo et al, 2016).
    3) NO EVIDENCE OF ZIKA VIRUS OR DENGUE VIRUS INFECTION: Prenatal ultrasound to evaluate for fetal abnormalities consistent with congenital Zika virus syndrome. If fetal abnormalities are present, repeat Zika virus rRT-PCR and IgM testing. Clinical management should be based on laboratory results. If fetal abnormalities are absent, prenatal care should be based on the potential ongoing risk of Zika virus exposure in pregnant women (Oduyebo et al, 2016).
    7) AMNIOTIC FLUID TESTING
    a) Amniotic fluid can be tested for the virus by RT-PCR. At present, the sensitivity and specificity to detect congenital infection remains unknown. It is also unknown if a positive test is predictive of a fetal abnormality (Petersen et al, 2016).
    E) POSTNATAL MONITORING OF WOMEN
    1) SUMMARY: Infants born to women that had tested positive for the Zika virus or possibly have the infection should be evaluated for congenital Zika virus infection. Zika virus testing is recommended for these infants regardless of the presence or absence of phenotypic abnormalities (Oduyebo et al, 2016).
    F) INFANTS
    1) BACKGROUND: The Zika virus infection has been transmitted through both intrauterine transmission (congenital) and intrapartum transmission from a viremic pregnant woman to her newborn. It has also been detected in breast milk (Zika virus RNA); however, transmission through breastfeeding has not been reported (Staples et al, 2016).
    2) Due to concerns about microencephaly associated with maternal exposure to the Zika virus, fetuses and infants of women infected should be evaluated for congenital exposure and possible neurologic abnormalities (Centers for Disease Control and Prevention (CDC), 2016). Currently, molecular and serologic testing are recommended by the CDC to diagnose congenital infection because of the uncertainty in determining which method is most reliable in this age group (Staples et al, 2016).
    3) In the US, interim guidelines have been established by the CDC for healthcare providers who care for infants born to women who may have been potentially exposed to the Zika virus either through travel or living in an area with Zika transmission during pregnancy (Staples et al, 2016). The following guidelines are recommended (Staples et al, 2016):
    a) Pediatricians should work closely with obstetricians to identify pregnant women or new mothers that were potentially exposed to the Zika virus.
    b) Fetal ultrasounds and maternal testing for Zika should be reviewed.
    4) Infants should be tested for Zika virus based on the following (Staples et al, 2016):
    1) Infants with microcephaly or intracranial calcifications born to women who traveled to or resided in an area with Zika virus transmission.
    2) Infants born to mothers with a positive or inconclusive laboratory testing for Zika virus.
    3) Infants with possible laboratory evidence of congenital Zika virus exposure should receive further clinical evaluation and treatment as indicated.
    4) Providers should contact their local state health department to coordinate testing of an infant (as an arboviral disease it is a reportable disease).
    5) METHOD OF COLLECTION
    a) RT-PCR TESTING: Obtain serum specimens from the umbilical cord or directly from the infant within 2 days of birth (Staples et al, 2016).
    b) OTHER STUDIES: Samples should also be collected and tested from the placenta at the time of delivery or CSF fluid (if collected for another reason) and include RT-PCR, IgM ELISA for Zika and dengue viruses on infant and maternal serum. In some cases, cross-reactivity may produce a false-positive result; therefore, further testing using the plaque reduction neutralization tests (PRNT) method can measure virus-specific neutralizing antibodies to rule out cross-reactivity antibodies from closely related flaviviruses (eg, dengue or yellow fever viruses) (Staples et al, 2016).
    c) IMMUNOHISTOCHEMICAL STAINING: This method may be considered to detect the Zika virus antigen on fixed placenta and umbilical cord tissues (Staples et al, 2016).
    6) RESULTS OF TESTING
    a) POSITIVE CONGENITAL INFECTION: The infant is considered congenitally infected with the virus, if Zika virus RNA or viral antigen are present in a sample (ie, amniotic fluid, placenta, umbilical cord). A positive finding also includes, Zika virus IgM antibodies with confirmatory neutralizing antibody titers that are greater than or equal to 4-fold higher than dengue virus neutralizing antibody titers in the infant serum or cerebrospinal fluid (Staples et al, 2016).
    1) A positive rRT-PCR or immunohistochemical staining on the placenta indicates the presence of maternal infection (Oduyebo et al, 2016).
    b) INCONCLUSIVE FINDINGS: Results are inconclusive if the Zika virus neutralizing antibody titers are less than 4-fold higher than the dengue virus (Staples et al, 2016).
    c) Positive and inconclusive test results should be reported to state and local health department for ongoing follow-up. Testing should also include a repeat hearing test at 6 months even if the initial study was normal (Staples et al, 2016).
    7) OTHER STUDIES
    a) Perform a retinal exam within the first month in infants with a possible congenital Zika virus infection (Staples et al, 2016).

Life Support

    A) Support respiratory and cardiovascular function.

Patient Disposition

    6.3.6) DISPOSITION/BITE-STING EXPOSURE
    6.3.6.1) ADMISSION CRITERIA/BITE-STING
    A) Hospital admission is uncommon; however, unstable patients or those with worsening symptoms should be admitted to the hospital. Those with severe symptoms (eg, neurologic abnormalities, Guillain-Barre syndrome) may require an ICU admission. Criteria for discharge include patients that are clinically improving and stable.
    6.3.6.2) HOME CRITERIA/BITE-STING
    A) Asymptomatic patients or patients with minimal symptoms may remain at home.
    6.3.6.3) CONSULT CRITERIA/BITE-STING
    A) Infectious disease physicians and/or intensivists may be consulted for medical advice. Pregnant women who have tested positive for the Zika virus should be referred to an infectious disease specialist with expertise in prenatal management (Petersen et al, 2016). Rheumatologists should be consulted in patients that develop severe joint swelling, arthritis and/or polyarthritis symptoms. Poison centers can aid treatment by serving as a public health resource by providing advice to the general public, and working with local and regional health departments.
    6.3.6.5) OBSERVATION CRITERIA/BITE-STING
    A) Patients with worsening symptoms that do not improve with over-the-counter medications (eg, analgesics, antipyretics) and basic home treatments should go to a healthcare facility for evaluation and treatment. Patients who are stable with improving symptoms may be sent home.

Monitoring

    A) Monitor vital signs and mental status. In most cases, clinical illness is mild; however, Guillain-Barre has been reported in cases of suspected Zika virus.
    B) Any patient who presents with an acute febrile illness, rash, myalgia, or arthralgia and have travelled recently (within the past 2 weeks) where there is ongoing infection of the Zika virus or lives in an at-risk region should be tested. Testing should include Zika, chikungunya and dengue virus infections.
    C) DIAGNOSTIC EVALUATION: The diagnosis of the Zika virus is by molecular and serologic testing and includes reverse transcription-polymerase chain reaction (RT-PCR) for viral RNA, immunoglobulin M ELISA (IgM ELISA) and plaque reduction neutralization test (PRNT) for Zika antibodies. Diagnosis can vary depending on the stage of illness (ie, acute versus convalescent period). In the acute phase (first 7 days of illness), viral RNA can usually be identified in serum, and RT-PCR is preferred to test all 3 suspected types (Zika, dengue, chikungunya) of virus. In the convalescent period (serum collected more than 8 days after illness), patients should be tested by virus-specific IgM ELISA; positive results are confirmed by testing for neutralizing antibodies. For patients with a positive IgM (ie, dengue or Zika IgM ELISA), the results are considered indicative of a recent flavivirus infection; therefore, plaque-reduction neutralization (PRNT) tests should be performed to measure virus-specific neutralizing antibodies to possibly discriminate between cross-reacting antibodies in primary flavivirus infections.
    D) PREGNANT WOMEN: All pregnant women in the United States and US territories should be assessed for possible Zika virus exposure during each prenatal visit. Pregnant women who report signs or symptoms consistent with Zika virus exposure (ie, acute onset of fever, rash, arthralgia, conjunctivitis) should be tested for the virus.
    1) SYMPTOMATIC PREGNANT WOMEN: rRT-PCR testing of serum and urine should be performed in women that seek care less than 2 weeks after symptom onset. A positive result confirms the diagnosis of recent maternal Zika virus infection. Symptomatic pregnant women with a negative result should have Zika virus IgM and dengue virus IgM antibody testing performed. If either test are positive or equivocal results, PRNT should be performed on the same IgM-tested sample to rule out a false-positive result.
    2) Symptomatic pregnant women who seek care 2 to 12 weeks after symptom onset should initially have Zika virus and dengue virus IgM antibody testing done.
    3) In symptomatic and asymptomatic pregnant women with possible Zika virus exposure who seek care greater than 12 weeks after symptom onset or possible exposure, IgM antibody testing should be considered. Because of the limitations of testing beyond 12 weeks, serial fetal ultrasounds should be considered as indicated.
    4) ASYMPTOMATIC PREGNANT WOMEN: Testing of asymptomatic pregnant women differs depending on the possible circumstances of exposure. Asymptomatic and NOT living in an area with active Zika virus transmission less than 2 weeks after possible exposure: Perform rRT-PCR (serum and urine) testing. Asymptomatic and NOT living in an area with active Zika virus transmission 2 to 12 weeks after possible exposure OR asymptomatic and living in an area with active Zika transmission (includes first and second trimester): Obtain Zika and dengue IgM (serum).
    5) PRENATAL MONITORING OF POSITIVE/PROBABLE CASES: RECENT ZIKA VIRUS INFECTION: Consider serial ultrasounds every 3 to 4 weeks to follow fetal anatomy and growth. Amniocentesis is based on individual clinical circumstances. PRESUMPTIVE RECENT ZIKA VIRUS INFECTION: Consider serial ultrasounds every 3 to 4 weeks to follow fetal anatomy and growth. Amniocentesis is based on individual clinical circumstances. NO EVIDENCE OF ZIKA VIRUS OR DENGUE VIRUS INFECTION: Prenatal ultrasound to evaluate for fetal abnormalities consistent with congenital Zika virus syndrome. If fetal abnormalities are present, repeat Zika virus rRT-PCR and IgM testing. Clinical management should be based on laboratory results.
    E) INFANTS: Due to concerns about microencephaly associated with maternal exposure to the Zika virus, fetuses and infants of women infected with the virus should be evaluated for congenital exposure and possible neurologic abnormalities. Currently, molecular and serologic testing are recommended by the CDC to diagnose congenital infection because of the uncertainty in determining which method is most reliable in this age group.
    F) Monitor fluid status and electrolytes in patients that develop significant vomiting and/or diarrhea.
    G) No specific laboratory studies are needed for most patients that develop mild clinical symptoms. Obtain a baseline CBC in patients as indicated; the presence of neutropenia, thrombocytopenia and hemorrhage are not typical of Zika virus, but are more likely to occur with dengue virus infection.
    H) Beginning in 2016, the US FDA has issued an Emergency Use Authorization for several diagnostic tools that can be used for testing for the Zika virus. They include Trioplex Real Time RT-PCR assay and the Zika MAC-ELISA, which have been distributed to qualified laboratories in the US.
    I) BLOOD DONATION: As of late August, 2016 the US FDA has recommended that all blood donations (blood and blood components) collected in the US and US territories be tested for the Zika virus using an investigational individual donor nucleic acid test (ID-NAT). Donors that are reactive for Zika virus should be deferred from donating for 120 days from the date of the reactive test. Donors should also be counseled regarding a possible exposure/infection.

Summary

    A) TOXICITY: The Zika virus is primarily transmitted by the bite of an infected Aedes aegypti mosquitoes which is the same mosquito that can spread dengue and chikungunya viruses. During outbreaks of the Zika virus, anthroponotic (human-to-vector-to human) transmission occurs. Other modes of transmission have included perinatal, in utero, and potentially blood transfusions and sexual contact with an infected partner. The virus typically results in no symptoms or a mild self-limited illness. A specific viral dose is unknown. Fatalities are rare, but microencephaly has occurred in infants during the recent outbreak of the virus in Brazil. Other potentially serious adverse effects have been sporadic reports of Guillain-Barre syndrome in adults.

Minimum Lethal Exposure

    A) Fatalities have been rarely associated with Zika virus (Petersen et al, 2016; Centers for Disease Control and Prevention (CDC), 2016).

Maximum Tolerated Exposure

    A) PRIMARY EXPOSURE: The Zika virus is primarily transmitted through the bite of an infected Aedes aegypti mosquitoes, which is the same mosquito that can spread dengue and chikungunya viruses. During outbreaks of the Zika virus, anthroponotic (human-to-vector-to human) transmission can occur. Other modes of transmission have included perinatal, in utero, and potentially blood transfusions and sexual contact. The Zika virus has been detected in breast milk, but transmission of the virus through breastfeeding to date has not been reported (Centers for Disease Control and Prevention (CDC), 2016; Centers for Disease Control and Prevention (CDC), 2016; Staples et al, 2016).
    B) In February 2016, the first confirmed case of Zika virus acquired through sexual transmission was reported in Dallas. The patient was infected with the virus after having sexual contact with an ill partner who had returned from a country where the Zika virus was present (Dallas County Department of Health and Human Services, 2016). As of July 2016, 15 cases of Zika virus infection through sexual transmission have been reported in the United States (Brooks et al, 2016).
    C) Only 1 in 5 people infected with the Zika virus will become symptomatic. Symptoms usually last for several days to a week. Of the patients that may become ill, severe disease requiring hospitalization is uncommon. The virus can remain in the blood of an infected person for several days but it may be longer (Centers for Disease Control and Prevention (CDC), 2016).
    1) SEVERE ILLNESS: Guillain-Barre syndrome has been associated with some cases of Zika virus infection (Centers for Disease Control and Prevention (CDC), 2016).
    2) INFANTS: During an outbreak that started in 2015 in Brazil, microencephaly has been reported in some infants born to pregnant women exposed to the Zika virus. The association between pregnant women infected with the Zika virus and infants born with microencephaly or neurologic abnormalities is being studied (Staples et al, 2016).

General Bibliography

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